David Reeves:
CLASS OF 2011
Eastside High SchoolClass of 2011
Lancaster, CA
New Vista Middle SchoolClass of 2007
Lancaster, CA
Jonesboro Middle SchoolClass of 2007
Jonesboro, GA
Endeavour Middle SchoolClass of 2006
Lancaster, CA
Lincoln Elementary SchoolClass of 2004
Lancaster, CA
David's Story
Born December 27, 1992.
Birth City: Inglewood, CA
Birth Hospital: Dan Freeman Memorial Hospital.
Birth Time: 05:18 P.M. PDT
Los Angeles, CA
(1992-1999)
Riverside, CA
(1999)
Lancaster, CA
1999-2023 (Present)
Jonesboro, GA
Dec. 2006 - June 2007
Eastside High School
Lancaster, CA
2007-2011
Jonesboro Middle School
Jonesboro, GA
Dec. 2006 - June 2007
New Vista Middle School
Lancaster, CA
Jun. 2006 - Dec. 2006
Endeavour Middle School
Lancaster, CA
Aug. 2004 - May 2006
Lincoln Elementary School
Lancaster, CA
Aug. 1999 - June 2004
Wilshire Crest Elementary School
Los Angeles, CA
Aug. 1998 - 1999
Marlton Charter School
Los Angeles, CA
1996-1998
Antelope Valley College
Lancaster, CA
2013-2017
Animal Lover
Beach lover
4 Siblings
5’4” / Pudgy
Status: Single
Here For: Networking, Pen Pal’ing.
Sexual Orientation: Straight
Hometown: Los Angeles, CA
Current City: Lancaster, CA
Body Type: 5’4” / Pudgy
Ethnicity: African American
Religion: Agnostic Theist
Zodiac Sign: Capricorn
Education: High School
Occupation: Admissions Host / Ticket Taker.
Rest In Peace William Clay Settle
Oct. 18, 1993 - Aug. 13, 2016
Rest In Peace Mylik Leevon Wright
Jul. 3, 1995 - Jan. 6, 2022
Rest In Peace My’ckquan Eric Pleasant
Jul. 17, 1992 - May 31, 2022
Aniridia Awareness:
(A-nih-RIH-dee-uh) Aniridia is the absence of the iris, a muscular structure that opens and closes the pupil to allow light into the eyes. It is also responsible for eye color. Without it, the central eye appears all black. It can be congenital, in which both eyes are usually involved, or caused by a penetrant injury. Isolated aniridia is a congenital disorder which is not limited to a defect in iris development, but is a panocular condition with macular and optic nerve hypoplasia, cataract, and corneal changes. Vision may be severely compromised and the disorder is frequently associated with a number of ocular complications: nystagmus, amblyopia, buphthalmos, and cataract. Aniridia in some individuals occurs as part of a syndrome, such as WAGR Syndrome (Kidney nephroblastoma (Wilms tumor), genitourinary anomalies and intellectual disability), or Gillespie syndrome (cerebellar ataxia).
Symptoms
Aniridia can cause many symptoms, such as:
1) Glaucoma: Glaucoma is the result of increased pressure within the eye, which, if not treated, can permanently damage the optic nerve and cause vision loss. Juvenile onset glaucoma is frequent in aniridia patients, whereas it tends to occur in older people in the general population. Therefore, testing for glaucoma should begin at birth.
2) Cataracts: Cataracts are areas of clouding in the lens of the eye and occur in 50-85% of aniridia patients. Juvenile cataracts are common in aniridia patients but should only be removed surgically if they interfere with usable vision.
3) Strabismus: Strabismus is a misalignment of the eyes, where an eye “wanders” when looking straight at an object. This can be corrected if addressed early with either surgery or patching.
4) Amblyopia: Amblyopia, also called “lazy eye”, occurs when the brain does not fully acknowledge the vision being sent to it by one eye. Anything that interferes with clear vision in the eye during the first 6 years of life can create an amblyopic eye, including strabismus, ptosis, or cataract. Treatment early in life is important to strengthen vision.
5) Ptosis: Ptosis is an abnormal drooping of the upper eyelid. If the ptosis is severe enough to block vision, it can be treated with surgery.
6) Dry Eye: Poor quality tears are associated with aniridia, which can cause dry eyes...Expand for more
. Dry eyes can, in turn, aggravate the corneal problems observed in Aniridia. Preservative-free artificial tears can help to keep the eye moist, as can autologous serum eye drops.
7) Corneal Keratopathy: In a normal eye, the cornea is a clear covering over the pupil and iris. In up to 90% of aniridia patients, corneal abnormalities such as clouding and an overgrowth of blood vessels can occur. These abnormalities are called aniridia-associated keratopathy (AAK) Although this begins early in life, it typically doesn’t affect vision until adulthood.
8) Photophobia: Photophobia is increased light sensitivity, due to the lack of iris in the eye. Although indoor lighting is not harmful to someone with aniridia, it is common for some lights to be more comfortable than others. Bright sunlight can often be uncomfortable for people with aniridia, so sunglasses and hats are recommended even for infants and young children.
9) Nystagmus: Nystagmus is the term for involuntary eye movements. Sometimes referred to as “wobbly eyes”, it is believed that nystagmus is the result of foveal hypoplasia. Nystagmus in aniridia typically presents itself within the first 2-3 months of life, if not at birth.
10) Hypoplasia: Hypoplasia means “underdevelopment” of a tissue or organ. People with aniridia frequently have foveal or optic nerve hypoplasia. The fovea is the very center of the retina and is responsible for most detailed vision. The optic nerve carries the messages from the eyes to the brain. Hypoplasia is a frequent cause of baseline visual impairment in aniridia and cannot be corrected with glasses.
11) Aniridic Fibrosis: Aniridic Fibrosis Syndrome (AFS) is a potential complication resulting from surgery in an aniridic eye. Fibrous tissue forms in the anterior chamber of the eye, causing vision loss. Surgical intervention is often required when AFS is present.
Question: What is considered legally blind?
Answer: In order to be legally blind, you must have a visual acuity of 20/200. This means that EVEN WITH glasses or contacts, you can only read the first letter at the top of the Snellen Chart, if that. You can also be legally blind if you can see, but only in a very small window in your eye.
Question: How rare is aniridia?
Answer: This is a very rare disorder, occurring in one of 60,000-90,000 live births. Aniridia is inherited as a dominant gene, and therefore each child born to a parent with the condition will have a 50% chance of having the gene. Aniridia can be either congenital or acquired. Congenital aniridia is a rare disease that affects both eyes. It is autosomal dominant in the majority. Most of the other cases of congenital aniridia are sporadic.
Question: What is aniridia caused by?
Answer: Molecular and Genetic Basis of Aniridia. Isolated aniridia is caused by heterozygous mutations in the paired box gene-6 (PAX6; 607108) on chromosome 11p13 or deletion of a regulatory region controlling its expression. Pax genes are a family of developmental genes that encode nuclear transcription factors.
Question: Is there a cure for aniridia?
Answer: With careful treatment, children with aniridia will have a very good outcome although there is no cure and they will need to have assistance for adapting to living with reduced vision. This is a very rare disorder, occurring in one of 60,000-90,000 live births. An eye care provider may also treat aniridia by surgically replacing the iris with an artificial iris. This procedure for both adults and children may help reduce light sensitivity and glare. It may also improve how the eye looks.
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